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- W2100124602 abstract "Hypersensitivity pneumonitis (HP) is an immunologically-mediated lung disease caused by repeated inhalation of dispersed antigen. Various cytokines have been reported to be involved in the immunopathogenesis of HP. Recently, some reports suggested an association between the genetic control of cytokine production and disease susceptibility. To evaluate whether cytokine gene polymorphisms are associated with HP, we performed a case-control association study involving 61 patients with HP, consisting of summer-type HP (SHP) and bird fancier’s lung (BFL, also named bird fancier’s disease), as well as 101 healthy controls. Polymorphisms of the genes for tumor necrosis factor (TNF)-I (－ 308G/A, －857C/T, －863C/A, －1031T/C), interleukin (IL)-10 (－ 592C/A, － 819C/T, － 1082G/A), transforming growth factor (TGF)-C1 (－509C/T, ＋869T/C), and IL6 (－634C/G) were examined by restriction fragment length polymorphism analysis. There were no significant differences in allele frequency and genotype distribution among control, SHP, and BFL group. When HP group was divided into acute or chronic, no significant differences were detected between any groups. LPS-stimulated IL-6 secretion by whole blood cells was similar between subjects with GG genotype and non-GG genotype in IL-6 －634C/G polymorphism." @default.
- W2100124602 created "2016-06-24" @default.
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- W2100124602 date "2006-03-01" @default.
- W2100124602 modified "2023-09-25" @default.
- W2100124602 title "Proinflammatory and anti-inflammatory cytokine gene polymorphisms in hypersensitivity pneumonitis." @default.
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- W2100124602 doi "https://doi.org/10.11480/jmds.530110" @default.
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