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- W2100190218 endingPage "1727" @default.
- W2100190218 startingPage "1717" @default.
- W2100190218 abstract "Throughout the past decade, there have been substantial advances in understanding the pathogenesis of idiopathic pulmonary fibrosis (IPF). Recently, several large genome-wide association and linkage studies have identified common genetic variants in more than a dozen loci that appear to contribute to IPF risk. In addition, family-based studies have led to the identification of rare genetic variants in genes related to surfactant function and telomere biology, and mechanistic studies suggest pathophysiological derangements associated with these rare genetic variants are also found in sporadic cases of IPF. Current evidence suggests that rather than existing as distinct syndromes, sporadic and familial cases of IPF (familial interstitial pneumonia) probably reflect a continuum of genetic risk. Rapidly evolving bioinformatic and molecular biology techniques, combined with next-generation sequencing technologies, hold great promise for developing a comprehensive, integrated approach to defining the fundamental molecular mechanisms that underlie IPF pathogenesis." @default.
- W2100190218 created "2016-06-24" @default.
- W2100190218 creator A5001488233 @default.
- W2100190218 creator A5063872240 @default.
- W2100190218 creator A5065762639 @default.
- W2100190218 date "2015-04-02" @default.
- W2100190218 modified "2023-10-18" @default.
- W2100190218 title "The genetic basis of idiopathic pulmonary fibrosis" @default.
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