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- W2100222319 endingPage "3417" @default.
- W2100222319 startingPage "3406" @default.
- W2100222319 abstract "Abstract As the risks of allogeneic blood transfusion (ABT)–transmitted viruses were reduced to exceedingly low levels in the US, transfusion-related acute lung injury (TRALI), hemolytic transfusion reactions (HTRs), and transfusion-associated sepsis (TAS) emerged as the leading causes of ABT-related deaths. Since 2004, preventive measures for TRALI and TAS have been implemented, but their implementation remains incomplete. Infectious causes of ABT-related deaths currently account for less than 15% of all transfusion-related mortality, but the possibility remains that a new transfusion-transmitted agent causing a fatal infectious disease may emerge in the future. Aside from these established complications of ABT, randomized controlled trials comparing recipients of non–white blood cell (WBC)–reduced versus WBC-reduced blood components in cardiac surgery have documented increased mortality in association with the use of non-WBC–reduced ABT. ABT-related mortality can thus be further reduced by universally applying the policies of avoiding prospective donors alloimmunized to WBC antigens from donating plasma products, adopting strategies to prevent HTRs, WBC-reducing components transfused to patients undergoing cardiac surgery, reducing exposure to allogeneic donors through conservative transfusion guidelines and avoidance of product pooling, and implementing pathogen-reduction technologies to address the residual risk of TAS as well as the potential risk of the next transfusion-transmitted agent to emerge in the foreseeable future." @default.
- W2100222319 created "2016-06-24" @default.
- W2100222319 creator A5014421997 @default.
- W2100222319 creator A5088908851 @default.
- W2100222319 date "2009-04-09" @default.
- W2100222319 modified "2023-10-11" @default.
- W2100222319 title "Transfusion-related mortality: the ongoing risks of allogeneic blood transfusion and the available strategies for their prevention" @default.
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- W2100222319 doi "https://doi.org/10.1182/blood-2008-10-167643" @default.
- W2100222319 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19188662" @default.
- W2100222319 hasPublicationYear "2009" @default.
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