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- W2100341715 abstract "Long-term potentiation (LTP) is a form of synaptic plasticity, which may be one of the events underlying learning and memory. LTP is triggered by brief, high-frequency stimulation of afferents, resulting in a long-lasting increase in the effectiveness of synaptic transmission (Bliss and Lomo, 1973; Bliss and Lynch, 1988; Brown et al., 1988; Matthies, 1989). Traditionally, LTP has been divided into two phases, the initiation phase and the maintenance phase. At present, three phases can be distinguished: (i) an initiation phase, including several seconds after tetanic stimulation when the events that trigger LTP begin; (ii) a transient phase, lasting about 30 min, during which there is a slow decay of potentiation, which can be evoked by local transmitter application without stimulating the presynaptic terminal; and (iii) a maintenance phase, which can last for hours (Kauer et al., 1988; Malinow et al., 1988a)." @default.
- W2100341715 created "2016-06-24" @default.
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- W2100341715 creator A5072191939 @default.
- W2100341715 date "1990-01-01" @default.
- W2100341715 modified "2023-09-25" @default.
- W2100341715 title "The Role of Protein Kinase C Substrate B-50 (GAP-43) in Neurotransmitter Release and Long-Term Potentiation" @default.
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- W2100341715 doi "https://doi.org/10.1007/978-1-4684-5769-8_38" @default.
- W2100341715 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/1981651" @default.
- W2100341715 hasPublicationYear "1990" @default.
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