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- W2100488521 endingPage "1542" @default.
- W2100488521 startingPage "1529" @default.
- W2100488521 abstract "Multi-drug chemoresistance remains one of the most common reasons for chemotherapy failure. The membrane transporter protein ABCG2/BCRP1 has been shown in vitro to effectively reduce the intracellular concentrations of several prominent anticancer chemotherapeutic agents such as mitoxantrone and doxorubicin. Intriguingly, cancer stem cells are known to be characterized by multi-drug chemoresistance. Taking into account that the ABCG2(+) subset of tumor cells are often enriched with cells with cancer stem-like phenotypes, it has been proposed that ABCG2 activity underlies the ability of cancer cells to regenerate post-chemotherapy. Furthermore, we also review evidence suggesting that tyrosine kinase inhibitors, including imatinib and gefitinib, are both direct and downstream inactivators of ABCG2 and, therefore, serve as candidates to reverse cancer stem cell chemoresistance and potentially target cancer stem cells." @default.
- W2100488521 created "2016-06-24" @default.
- W2100488521 creator A5067079844 @default.
- W2100488521 creator A5077592721 @default.
- W2100488521 date "2009-08-27" @default.
- W2100488521 modified "2023-10-02" @default.
- W2100488521 title "ABCG2: the key to chemoresistance in cancer stem cells?" @default.
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