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- W2100548723 abstract "Significance Our observations provide evidence to link epidemiological studies implicating stress-related processes with biological dysfunction in type 2 diabetes. The patterns of cardiovascular, inflammatory, neuroendocrine, and cholesterol responses exemplify the disturbances of reactivity and recovery predicted by the allostatic load model, including prolonged responses to stress due to delayed shutdown of physiological reactivity, and inadequate (blunted) responses resulting in compensatory hyperactivity in other mediating pathways. Chronic allostatic load may be a mechanism through which stress exposures contribute to diabetes risk, while also being implicated in the adverse health consequences of diabetes such as coronary heart disease and cognitive decline." @default.
- W2100548723 created "2016-06-24" @default.
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- W2100548723 date "2014-10-20" @default.
- W2100548723 modified "2023-10-15" @default.
- W2100548723 title "Disruption of multisystem responses to stress in type 2 diabetes: Investigating the dynamics of allostatic load" @default.
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- W2100548723 doi "https://doi.org/10.1073/pnas.1410401111" @default.
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