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- W2100806851 abstract "Platelets and their interaction with cells of the immune system contribute through a variety of molecular mechanisms to support hemostasis and inflammation. These simple yet essential cells exert their effects in lymphocytes, monocytes, and neutrophils, both recruiting and modulating their function after activation. Emerging evidence is starting to define the mechanisms that allow platelets to also play pivotal roles in host defense. For example, platelet cell-surface expression of toll-like receptors allows platelets to direct neutrophil activation toward extracellular trap formation and facilitate the elimination of blood pathogens. In addition to these well-known receptors, two of the most recently discovered platelet receptors, C-type lectin receptor 2 (CLEC-2), and TREM-like transcript-1 (TLT-1), have been shown to modulate hemostatic and inflammation-related roles in platelets. This review will discuss the evolution of our understanding of platelet functions from hemostasis to inflammation, and highlight novel mechanisms that platelets use to mediate hemostasis under inflammatory pressure." @default.
- W2100806851 created "2016-06-24" @default.
- W2100806851 creator A5002506109 @default.
- W2100806851 creator A5042227012 @default.
- W2100806851 creator A5059407347 @default.
- W2100806851 creator A5069920875 @default.
- W2100806851 date "2014-01-01" @default.
- W2100806851 modified "2023-10-18" @default.
- W2100806851 title "Emerging Roles for Platelets in Inflammation and Disease" @default.
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- W2100806851 doi "https://doi.org/10.4172/2332-0877.1000149" @default.
- W2100806851 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5531291" @default.
- W2100806851 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/28758142" @default.
- W2100806851 hasPublicationYear "2014" @default.
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