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- W2101476930 endingPage "274" @default.
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- W2101476930 abstract "The role of protein kinase C (PKCs) isoforms in the regulation of glucose metabolism by insulin is complex, partly due to the large PKC family consisting of three sub-groups: conventional, novel and atypical. Activation of some conventional and novel PKCs in response to increased levels of diacylglycerol (DAG) have been shown to counteract insulin signalling. However, roles of atypical PKCs (aPKCs) remain poorly understood. aPKCs act as molecular switches by promoting or suppressing signalling pathways, in response to insulin or ceramides respectively. Understanding how DAG- and ceramide-activated PKCs impair insulin signalling would help to develop treatments to fight insulin resistance." @default.
- W2101476930 created "2016-06-24" @default.
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- W2101476930 date "2010-12-19" @default.
- W2101476930 modified "2023-10-18" @default.
- W2101476930 title "Protein kinase C isoforms: Mediators of reactive lipid metabolites in the development of insulin resistance" @default.
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- W2101476930 doi "https://doi.org/10.1016/j.febslet.2010.12.022" @default.
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