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- W2102564339 endingPage "511" @default.
- W2102564339 startingPage "487" @default.
- W2102564339 abstract "In plant innate immunity, individual cells have the capacity to sense and respond to pathogen attack. Intracellular recognition mechanisms have evolved to intercept perturbations by pathogen virulence factors (effectors) early in host infection and convert it to rapid defense. One key to resistance success is a polymorphic family of intracellular nucleotide-binding/leucine-rich-repeat (NLR) receptors that detect effector interference in different parts of the cell. Effector-activated NLRs connect, in various ways, to a conserved basal resistance network in order to transcriptionally boost defense programs. Effector-triggered immunity displays remarkable robustness against pathogen disturbance, in part by employing compensatory mechanisms within the defense network. Also, the mobility of some NLRs and coordination of resistance pathways across cell compartments provides flexibility to fine-tune immune outputs. Furthermore, a number of NLRs function close to the nuclear chromatin by balancing actions of defense-repressing and defense-activating transcription factors to program cells dynamically for effective disease resistance." @default.
- W2102564339 created "2016-06-24" @default.
- W2102564339 creator A5013706725 @default.
- W2102564339 creator A5063094198 @default.
- W2102564339 creator A5078978940 @default.
- W2102564339 date "2015-04-29" @default.
- W2102564339 modified "2023-10-18" @default.
- W2102564339 title "Effector-Triggered Immunity: From Pathogen Perception to Robust Defense" @default.
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