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• W2102594860 abstract "The effects of bafilomycin A1 and of the reduced level of endosomal epsilon-COP (coatomer protein) on the infectivity of human adenovirus type 5 were investigated in Coxsackie adenovirus receptor- (CAR-) transfected Chinese hamster ovary (CHO) cells. The endosomal proton pump inhibitor bafilomycin A1 was able to cause only partial inhibition. Using Id1F cells (an epsilon-COP thermosensitive mutant CHO cell line) the reduction of epsilon-COP level also had partial inhibitory effect. Based on these results and comparing them to existing models of the adenovirus entry, we propose a refined model in which there are two pathways of adenoviral entry: the first one involves the epsilon-COP as the downstream effector of the acidification and can be blocked by bafilomycin A1 and the second one is a pH-independent pathway." @default.
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• W2102594860 date "2002-10-01" @default.
• W2102594860 modified "2023-09-25" @default.
• W2102594860 title "The endosomal epsilon-coatomer protein is involved in human adenovirus type 5 internalisation" @default.
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• W2102594860 doi "https://doi.org/10.1556/avet.50.2002.4.10" @default.
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