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• W2103127997 abstract "Summary Three Harwich P sublines with different P -element activity potential were used to investigate the influence of P-derived chromosomes on sn ω mutability and vg suppression and to relate the induction of these dysgenic traits to the number and structure of P elements. Destabilization of the sn ω allele, a measure of P transposase activity, was differentially influenced by the major autosomes. Chromosome 2 of the standard Harwich subline, H w , induced only 60% of the level of mutability relative to chromosome 3, whereas chromosome 3 of the weakest Harwich subline, H f , induced only 50% of the mutability relative to chromosome 2. In somatic suppression of the vg 21–3 allele, chromosome 3 of the H f subline produced a lower level of complete suppression as compared to chromosome 3 of the H w or the H s subline (the high hybrid-dysgenesis-inducing subline). The level of these dysgenic traits and GD sterility, was not correlated with the number of P elements per individual (67–68) or per chromosome arm which was very similar among the sublines. The number of complete P elements per genome, based on Southern blot analysis of the X and major autosomes, ranged from 15 to 19. Destabilization of the sn ω allele and vg suppression by chromosome 3 was correlated with a greater number of complete P elements. Two novel unexpected observations emerged from these studies: both sn ω mutability and vg suppression data demonstrated high P -element activity in hybrids derived from non-dysgenic crosses irrespective of Harwich subline, indicating a lack of P-cytotype regulation. Mutability in non-dysgenic males ranged from 40 to 60% of the level found in dysgenic males. The high sn ω mutability and low GD sterility in non-dysgenic hybrids suggests that these traits may arise by a different mechanism." @default.
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• W2103127997 date "1993-10-01" @default.
• W2103127997 modified "2023-09-27" @default.
• W2103127997 title "Mutability, sterility and suppression in P—M hybrid dysgenesis: the influence ofPsubline, cross, chromosome, sex and P-element structure" @default.
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• W2103127997 doi "https://doi.org/10.1017/s0016672300031700" @default.
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