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- W2103222799 abstract "In many genomes, toxin-antitoxin (TA) systems have been identified; however, their role in cell physiology has been unclear. Here we examine the evidence that TA systems are involved in biofilm formation and persister cell formation and that these systems may be important regulators of the switch from the planktonic to the biofilm lifestyle as a stress response by their control of secondary messenger 3',5'-cyclic diguanylic acid. Specifically, upon stress, the sequence-specific mRNA interferases MqsR and MazF mediate cell survival. In addition, we propose that TA systems are not redundant, as they may have developed to respond to specific stresses." @default.
- W2103222799 created "2016-06-24" @default.
- W2103222799 creator A5040949722 @default.
- W2103222799 creator A5082173877 @default.
- W2103222799 date "2011-08-15" @default.
- W2103222799 modified "2023-10-11" @default.
- W2103222799 title "Toxin-Antitoxin Systems Influence Biofilm and Persister Cell Formation and the General Stress Response" @default.
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- W2103222799 doi "https://doi.org/10.1128/aem.05068-11" @default.
- W2103222799 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3165247" @default.
- W2103222799 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21685157" @default.
- W2103222799 hasPublicationYear "2011" @default.
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