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• W2103263313 abstract "OBJECTIVE; ; The objective of this study was to identify developmental changes in vascular smooth muscle cells (VSMC) during aneurysm formation using an elastase-induced rabbit carotid artery aneurysm model.- Aneurysms were induced in carotid arteries of rabbits by exposure to elastase. Tissues from developing aneurysms were isolated at various time points for histological, immunohistochemical and RT-PCR analysis.During aneurysm growth, the vascular media became thinner and elastic fibres became fragmented.The expression of smooth muscle marker SM22alpha and cytoskeleton proteins (SM alpha-actin, beta-tubulin and desmin) expression was decreased while platelet-derived growth factor (PDGF) was increased, which correlated with a remodelling ofVSMC from contractile to synthetic functions.During the development of aneurysm, despite the induction of apoptosis, VSMC undergoes a remodelling from a contractile phenotype to a synthetic phenotype as a potential repair mechanism. Further studies may reveal the causative effects between these events." @default.
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• W2103263313 date "2010-10-01" @default.
• W2103263313 modified "2023-10-05" @default.
• W2103263313 title "Vascular smooth muscle cell remodelling in elastase-induced aortic aneurysm" @default.
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• W2103263313 doi "https://doi.org/10.1080/ac.65.5.2056235" @default.
• W2103263313 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/21125970" @default.
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