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- W2103282170 abstract "Recently, the notion that memory and addiction share similar neural substrates has become widely accepted. N-methyl-d-aspartate receptors (NMDAR) are the cornerstones of synaptic models of memory. The present study examined the effect of the competitive NMDAR antagonist CPP on the induction of behavioral sensitization and conditioned place preference to cocaine. Conditioned place preference is an associative memory model of drug seeking, while sensitization is a non-associative model of the transition from casual to compulsive use. There were three principal findings: (1) co-administration of CPP and cocaine altered the acute response to cocaine, suggesting a direct interaction between the two drugs; (2) NMDAR antagonism had no effect on behavioral sensitization; and (3) NMDAR antagonism abolished conditioned place preference. A review of prior evidence supporting a role for NMDARs in sensitization suggests that NMDAR antagonists directly interfere with cocaine's psychostimulant effects, and this interaction could be misinterpreted as a disruption of sensitization. Finally, we suggest that addiction recruits multiple kinds of plasticity, with sensitization recruiting NMDAR-independent mechanisms." @default.
- W2103282170 created "2016-06-24" @default.
- W2103282170 creator A5003744270 @default.
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- W2103282170 creator A5053888222 @default.
- W2103282170 creator A5082561813 @default.
- W2103282170 date "2013-02-01" @default.
- W2103282170 modified "2023-10-18" @default.
- W2103282170 title "The competitive NMDA receptor antagonist CPP disrupts cocaine-induced conditioned place preference, but spares behavioral sensitization" @default.
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- W2103282170 doi "https://doi.org/10.1016/j.bbr.2012.10.042" @default.
- W2103282170 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/23153931" @default.
- W2103282170 hasPublicationYear "2013" @default.