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- W2103290710 abstract "Initial reports of the presence of a specific mutation in the kinase domain of BCR-ABL[1][1] as the basis of clinical resistance as well as progression in chronic myeloid leukemia (CML) came on the heels of another first report of Abl kinase inhibitor success,[2][2] and the former was both a beacon" @default.
- W2103290710 created "2016-06-24" @default.
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- W2103290710 date "2013-04-30" @default.
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- W2103290710 title "T315I, more or less, predicts for major molecular response: the devil is in the details!" @default.
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- W2103290710 doi "https://doi.org/10.3324/haematol.2013.084251" @default.
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