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• W2103396305 startingPage "2431" @default.
• W2103396305 abstract "Diabetes results from an inadequate functional β cell mass, either due to autoimmune destruction (Type 1 diabetes) or insulin resistance combined with β cell failure (Type 2 diabetes). Strategies to enhance β cell regeneration or increase cell proliferation could improve outcomes for patients with diabetes. Research conducted over the past several years has revealed that factors regulating embryonic β cell mass expansion differ from those regulating replication of β cells post-weaning. This article aims to compare and contrast factors known to control embryonic and postnatal β cell replication. In addition, we explore the possibility that connective tissue growth factor (CTGF) could increase adult β cell replication. We have already shown that CTGF is required for embryonic β cell proliferation and is sufficient to induce replication of embryonic β cells. Here we examine whether adult β cell replication and expansion of β cell mass can be enhanced by increased CTGF expression in mature β cells." @default.
• W2103396305 created "2016-06-24" @default.
• W2103396305 creator A5005860428 @default.
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• W2103396305 creator A5049426852 @default.
• W2103396305 creator A5080973682 @default.
• W2103396305 date "2012-07-01" @default.
• W2103396305 modified "2023-10-11" @default.
• W2103396305 title "Differential regulation of embryonic and adult β cell replication" @default.
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• W2103396305 doi "https://doi.org/10.4161/cc.20545" @default.
• W2103396305 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3404874" @default.
• W2103396305 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/22659844" @default.
• W2103396305 hasPublicationYear "2012" @default.
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