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• W2104326307 abstract "Molecular mechanisms in the brain are assumed to cause the symptoms and severity of neuropsychiatric disorders. This review concerns the elusive nature of relationships between the severity of depressive disorders and neuromolecular processes studied by positron emission tomography (PET). Recent PET studies of human depression have focused on serotonergic, dopaminergic, muscarinic, nicotinic, and GABAergic receptors, as well as central processes dependent on monoamine oxidase, phosphodiesterase type 4, amyloid plaques, neurofibrillar tangles, and P-glycoprotein. We find that reliable causal links between neuromolecular mechanisms and relief from depressive disorders have yet to be convincingly demonstrated. This situation may contribute to the currently limited use of PET for exploring the neuropathways that are currently viewed as being responsible for beneficial effects of antidepressant treatment regimes." @default.
• W2104326307 created "2016-06-24" @default.
• W2104326307 creator A5080138773 @default.
• W2104326307 creator A5081485513 @default.
• W2104326307 date "2013-01-01" @default.
• W2104326307 modified "2023-09-25" @default.
• W2104326307 title "Molecular Neurobiology of Depression: PET Findings on the Elusive Correlation with Symptom Severity" @default.
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