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• W2104779106 abstract "As a class, nonsteroidal anti-inflammatory drugs (NSAIDs) are a leading cause of drug-induced liver disease. For instance, in Denmark, NSAIDs accounted for approximately 9% (97 of 1100) of all drug-related liver injury reports between 1978 and 1987.20 The early history of NSAID-induced hepatic injury dates back more than 60 years.89 Cinchophen was one of the earliest agents to be associated with hepatotoxicity. Its case fatality rate of nearly 50% forced its withdrawal from clinical use.54 Numerous other NSAIDs have been developed or introduced into practice only to be abandoned during premarket or postmarket evaluation because of serious liver injury.42 Examples of such agents include glafenine,78 an NSAID similar to cinchophen; ibufenac,14 a precursor of ibuprofen; fenclozic acid, an early arylalkanoic acid NSAID29 and fluproquazone, the precursor compound of quinazolone derivatives.46 However, it was not until the withdrawal of benoxaprofen in 1982 because of reports of fatal jaundice in the United Kingdom that attention was intensively focused on the hepatotoxicity of NSAIDs as a group.46 At that time, the Arthritis Advisory Committee of the Food and Drug Administration (FDA) concluded that hepatic injury should be considered a class characteristic of NSAIDs.55 However, this uniform characterization of hepatic injury obscures the many individual differences found within and among the different NSAID classes. The types and mechanisms of hepatic injury, as well as the hepatotoxic potential for different NSAID agents also can vary considerably. This article reviews the clinical-pathologic features of the hepatic injury associated with the currently available NSAIDs. Acetaminophen is not a true nonsteroidal anti-inflammatory drug and will not be discussed." @default.
• W2104779106 created "2016-06-24" @default.
• W2104779106 creator A5079409390 @default.
• W2104779106 date "1998-08-01" @default.
• W2104779106 modified "2023-09-25" @default.
• W2104779106 title "NSAID-INDUCED HEPATOTOXICITY" @default.
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• W2104779106 doi "https://doi.org/10.1016/s1089-3261(05)70026-x" @default.
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