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• W2105038229 abstract "Annals of the New York Academy of SciencesVolume 499, Issue 1 p. 124-131 Alterations in Nutrient Intake and Utilization Caused by Disease ROBERT A. HOERR, ROBERT A. HOERR Department of Applied Biological Sciences and the Clinical Research Center, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139Search for more papers by this authorVERNON R. YOUNG, VERNON R. YOUNG Department of Applied Biological Sciences and the Clinical Research Center, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139Search for more papers by this author ROBERT A. HOERR, ROBERT A. HOERR Department of Applied Biological Sciences and the Clinical Research Center, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139Search for more papers by this authorVERNON R. YOUNG, VERNON R. YOUNG Department of Applied Biological Sciences and the Clinical Research Center, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139Search for more papers by this author First published: June 1987 https://doi.org/10.1111/j.1749-6632.1987.tb36203.xCitations: 3AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL References 1 Gil, K. M., R. A. Forse, J. Askanazi, C. Weissman & J. M. Kinney. 1985. Energy metabolism in stress. In Substrate and Energy Metabolism. J. S. Garrow & D. Halliday, Eds.: 203– 211. J. Libbey. London . 2 Little, R. A. 1985. Heat production after injury. 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The glucose metabolism of patients with malignant disease and of normal subjects as studied by means of an intravenous glucose tolerance test. J. Clin. Invest. 36: 254– 264. 32 Lundholm, K., A.-C. Bylund & T. Schersten. 1977. Glucose tolerance in relation to skeletal muscle enzyme activities in cancer patients. Scand. J. Clin. Lab. Invest. 37: 267– 272. 33 Waterhouse, C., N. Jeanpretre & J. Keilson. 1979. Gluconeogenesis from alanine in patients with progressive malignant disease. Cancer Res. 39: 1968– 1972. 34 Waterhouse, C. 1974. Lactate metabolism in patients with cancer. Cancer (Brussels) 33: 66– 71. 35 Lundholm, K., S. Edstrom, L. Ekman et al. 1978. A comparative study of the influence of malignant tumor on host metabolism in mice and man. Cancer (Brussels) 42: 453– 461. 36 Lundholm, K., S. Edstrom, L. Ekman, I. Karlberg & T. Schersten. 1981. Metabolism in peripheral tissues in cancer patients. Cancer Treat. Rep. 65 (suppl. 5): 79– 83. 37 Weber, G. 1977. Enzymology of cancer cells (first of two parts). N. Eng. J. Med. 296: 486– 493. 38 Weber, G. 1977. Enzymology of cancer cells (second of two parts). N. Eng. J. Med. 296: 541– 551. 39 Murray, M. J. & A. B. Murray. 1979. Anorexia of infection as a mechanism of host defense. Am. J. Clin. Nutr. 32: 593– 596. 40 Dinarello, C. A. 1984. Interleukin-1 and the pathogenesis of the acute-phase response. N. Engl. J. Med. 311: 1413– 1418. 41 McCarthy, D. O., M. J. Kluger & A. J. Vander. 1985. Suppression of food intake during infection: is interluekin-1 involved? Am. J. Clin. Nutr. 42: 1179– 1182. 42 Wolfe, R. R. 1985. Glucose metabolism in burn injury. J. Burn Care Rehabil. 6: 408– 418. 43 Shaw, J. H. F., S. Klein & R. R. Wolfe. Assessment of alanine, urea and glucose interrelationships in normal subjects and in patients with sepsis with stable isotope tracers. Surgery 97: 557– 567. 44 Wolfe, R. R., M. J. Durkot, J. R. Allsop et al. 1979. Glucose metabolism in severely burned patients. Metab. Clin. Exp. 28: 1031– 1039. 45 Wolfe, R. R., T. F. O'Donnell, M. D. Stone, D. A. Richmond & J. F. Burke. 1980. Investigation of factors determining the optimal glucose infusion rate in total parenteral nutrition. Metab. Clin. Exp. 29: 892– 900. 46 Lund P. & D. H. Williamson. 1985. Inter-tissue nitrogen fluxes. Br. Med. Bull. 41: 251– 256. 47 Gelfand, R. A., R. A. DeFronzo & R. Gusberg. 1983. Metabolic alterations associated with major injury and infection. In New Aspects of Clinical Nutrition. G. Kleinberger & E. Dentach, Eds.: 211– 239. Karger. Basel . 48 Editorial. 1985. Cachectin. Lancet 2: 312– 313. 49 Pekala, P. H., S. R. Price, C. A. Horn, B. E. Hom, J. Moss & A. Cerami. 1984. Model for cachexia in chronic disease: secretory products of endotoxin-stimulated macrophages induce a catabolic state in 3T3-L1 adipocytes. Trans. Assoc. Am. Physicians 97: 251– 259. 50 Matthews, N. 1983. Effect on human monocyte killing of tumour cells of antibody raised against an extracellular monocyte cytotoxin. Immunology 48: 321– 327. 51 Beutler, B., D. Greenwald, J. D. Holmes, M. Chang, U.-C. E. Pan, J. Mathison, R. Ulevitch & A. Cerami. 1985. Identity of tumor necrosis factor and the macrophage-secreted factor cachectin. Nature (London) 316: 552. Citing Literature Volume499, Issue1Human ObesityJune 1987Pages 124-131 ReferencesRelatedInformation" @default.
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