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- W2105087807 abstract "Chronic thromboembolic pulmonary hypertension (CTEPH) is a life-threatening disease due to pulmonary artery obstruction by persistent organised clots related to one or more episodes of acute pulmonary embolism. To date, the pathogenesis of CTEPH remains unexplained. Pulmonary endarterectomy removes obstruction from pulmonary vessels and can cure patients. However, some unreachable distal pulmonary obstruction and/or associated distal pulmonary vasculopathy could induce persistent pulmonary hypertension, the main postoperative complication. The pathophysiology of CTEPH is not fully understood and improving knowledge of this disease could improve our future surgical and medical management. Many attempts, conducted over several decades, have failed to reproduce this chronic disease in animals. However, several animal models have provided insights into the pathophysiology and pathogenesis of CTEPH. Here, we review all the animal models that have improved the comprehension of CTEPH and hold promise for further investigations. This short review analyses strengths and weaknesses of all animal models available to study the pathophysiology of CTEPH." @default.
- W2105087807 created "2016-06-24" @default.
- W2105087807 creator A5029738332 @default.
- W2105087807 creator A5085705865 @default.
- W2105087807 date "2013-01-11" @default.
- W2105087807 modified "2023-10-14" @default.
- W2105087807 title "Chronic thromboembolic pulmonary hypertension: animal models" @default.
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- W2105087807 doi "https://doi.org/10.1183/09031936.00101612" @default.
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