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• W2105135621 abstract "Mutations of GPCRs can increase their constitutive (agonist-independent) activity. Some of these mutations have been artificially introduced by site-directed mutagenesis; others occur spontaneously in human diseases. The analysis of constitutively active GPCR mutants has attracted a large interest in the past decade, providing an important contribution to our understanding of the molecular mechanisms underlying receptor function and drug action." @default.
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• W2105135621 date "2003-04-01" @default.
• W2105135621 modified "2023-10-16" @default.
• W2105135621 title "Constitutively Active G Protein-Coupled Receptor Mutants: Implications on Receptor Function and Drug Action" @default.
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• W2105135621 doi "https://doi.org/10.1089/15406580360545125" @default.
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