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• W2105267515 startingPage "1331" @default.
• W2105267515 abstract "Abstract HLA-G is involved in regulating T cell responses. Various mechanisms have been proposed to explain the inhibition of T cell proliferation. In this context, the possible role of HLA-G in cell cycle regulation remains to be explored. Using stably transfected M8 cells expressing the secreted isoform (HLA-G5) of HLA-G, we investigated the role of HLA-G in inducing apoptosis and in controlling the cell cycle of activated T cells. Soluble HLA-G (HLA-G5) inhibited both CD4 and CD8 T cell proliferation. However, HLA-G5 did not induce T cell apoptosis, as determined by 3,3′-diethyloxacarbocyanine and propidium iodine labeling. It induced accumulation of the retinoblastoma protein, but not its phosphorylated and active form. Treatment of activated T cells with HLA-G5 also reduced the amounts of cyclin D2, E, A, and B by &gt;80%. In contrast, it induced an accumulation of p27kip, but not p21cip, in activated T cells. HLA-G does not induce apoptosis of alloreactive T cells, but induces p27kip1 and inhibits cell cycle progression." @default.
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• W2105267515 date "2006-02-01" @default.
• W2105267515 modified "2023-09-27" @default.
• W2105267515 title "Soluble HLA-G Inhibits Cell Cycle Progression in Human Alloreactive T Lymphocytes" @default.
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• W2105267515 doi "https://doi.org/10.4049/jimmunol.176.3.1331" @default.
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