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• W2105651504 endingPage "1348" @default.
• W2105651504 startingPage "1341" @default.
• W2105651504 abstract "c-Jun N-terminal kinases (JNKs), also referred to as stress-activated kinases (SAPKs), were initially characterized by their activation in response to cell stress such as UV irradiation. JNK/SAPKs have since been characterized to be involved in proliferation, apoptosis, motility, metabolism and DNA repair. Dysregulated JNK signaling is now believed to contribute to many diseases involving neurodegeneration, chronic inflammation, birth defects, cancer and ischemia/reperfusion injury. In this review, we present our current understanding of JNK regulation and their involvement in homeostasis and dysregulation in human disease." @default.
• W2105651504 created "2016-06-24" @default.
• W2105651504 creator A5043895289 @default.
• W2105651504 creator A5047612906 @default.
• W2105651504 date "2007-08-01" @default.
• W2105651504 modified "2023-10-14" @default.
• W2105651504 title "The c-jun kinase/stress-activated pathway: Regulation, function and role in human disease" @default.
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