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- W2107132783 abstract "SUMMARY: Secondary hyperparathyroidism is an adaptive response to progressive loss of renal function so as to maintain calcium and phosphate homeostasis, 1,25-dihydroxyvitamin D3 levels and normal bone turnover, despite skeletal resistance to parathyroid hormone. As feedback regulation fails, complications of parathyroid overactivity develop, and by the commencement of dialysis abnormal bone histology is present in almost all patients, with hyperparathyroid changes most commonly found. Post transplantation, persisting hyperparathyroidism predisposes to osteoporosis. The risk of bone disease is reduced by early, carefully targeted dietary measures and suppressive therapy with calcitriol and calcium-based phosphate binders, while newer therapies include bisphosphonates and calcimimetics. Timely surgical intervention is necessary in some patients." @default.
- W2107132783 created "2016-06-24" @default.
- W2107132783 creator A5025398073 @default.
- W2107132783 date "2001-08-01" @default.
- W2107132783 modified "2023-10-14" @default.
- W2107132783 title "Pathogenesis and management of hyperparathyroidism in end-stage renal disease and after renal transplantation" @default.
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- W2107132783 doi "https://doi.org/10.1046/j.1440-1797.2001.00038.x" @default.
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