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• W2107266724 startingPage "349" @default.
• W2107266724 abstract "Diabetes exacerbates neuronal cell death induced by cerebral ischemia. One contributing factor is enhanced acidosis during ischemia. Astrocytes are vulnerable to hypoxia under acidic conditions in vitro and may be targets of ischemia under diabetic conditions. The objective of this study was to determine whether diabetes would cause damage to astrocytes after an ischemic brain injury in vivo. Diabetic and nondiabetic rats were subjected to 5 min of forebrain ischemia and followed by 30 min, 6 h, or 1 or 3 days of recovery. The results showed that ischemia caused activation of astrocytes in nondiabetic rats. In contrast, diabetes caused astrocyte activation in early stage of reperfusion and astrocyte death in late stage of reperfusion. Remarkable astrocyte death was preceded by increased DNA oxidation. Further studies revealed that increased astrocyte damage coincided with enhanced production of free radicals. These data suggest that hyperglycemic ischemia worsens outcome in astrocytes, as it does in neurons." @default.
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• W2107266724 date "2006-02-01" @default.
• W2107266724 modified "2023-10-14" @default.
• W2107266724 title "Streptozotocin-Induced Diabetes Causes Astrocyte Death After Ischemia and Reperfusion Injury" @default.
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• W2107266724 doi "https://doi.org/10.2337/diabetes.55.02.06.db05-0654" @default.
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