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- W2108108520 abstract "ALTHOUGH IT IS well established that the adrenalectomized animal is hypersensitive to vascular stress, the compensatory mechanisms which fail in the absence of adreno-cortical hormones have not been directly demonstrated. In normal intact animals, it is known that resistance to hemorrhage, as well as to other procedures which reduce the blood volume, is dependent in great part upon emergency reflex mechanisms which operate to maintain venous return and cardiac output. The efficiency of these compensatory adjustments may be appreciated from the finding that hemorrhage equal to 2.1 per cent of the body weight may not decrease venous return and cardiac output at all (Meek and Eyster, 1921), presumably because reserve blood from less active regions of the circulation is shifted to the active circulation. The present work was undertaken to determine whether the hypersensitivity of adrenalectomized animals could be due to a primary failure of compensatory mechanisms for maintenance of venous return and cardiac ..." @default.
- W2108108520 created "2016-06-24" @default.
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- W2108108520 date "1945-02-01" @default.
- W2108108520 modified "2023-09-27" @default.
- W2108108520 title "CONCERNING THE HYPERSENSITIVITY OF ADRENALECTOMIZED RATS TO VASCULAR STRESS1" @default.
- W2108108520 doi "https://doi.org/10.1210/endo-36-2-77" @default.
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