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- W2108196559 endingPage "714" @default.
- W2108196559 startingPage "702" @default.
- W2108196559 abstract "A number of studies have confirmed the potential for neuronal nicotinic acetylcholine receptor (NNR)-mediated neuroprotection and, more recently, its anti-inflammatory effects. The mechanistic overlap between these pathways and the ubiquitous effects observed following diverse insults suggest that NNRs modulate fundamental pathways involved in cell survival. These results have wide-reaching implications for the design of experimental therapeutics that regulate inflammatory and anti-apoptotic responses through NNRs and represent an initial step toward understanding the benefits of novel therapeutic strategies for the management of central nervous system disorders that target neuronal survival and associated inflammatory processes." @default.
- W2108196559 created "2016-06-24" @default.
- W2108196559 creator A5004880516 @default.
- W2108196559 date "2009-06-01" @default.
- W2108196559 modified "2023-10-06" @default.
- W2108196559 title "Neuronal nicotinic receptors as novel targets for inflammation and neuroprotection: mechanistic considerations and clinical relevance" @default.
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