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- W2108422911 abstract "A 37-year-old man with a 15-year history of symptomatic, drugrefractory paroxysmal atrial fibrillation (AF) was referred to our hospital for AF ablation [1–4]. He had no structural heart disease, and the echocardiographic parameters were normal. The procedure was performed under conscious sedation. A 7-F 14 pole two site mapping catheter (Irvine Biomedical Inc., Irvine, CA, USA) was inserted through the right jugular vein and positioned in the coronary sinus for pacing, recording and internal cardioversion. Upon arrival, the patient was in sinus rhythm, but AF was easily induced by burst pacing from the right atrium. We performed several attempts of cardioversion (maximal external cardioversion delivery of 360 J, and maximal internal cardioversion delivery of 10 J) to find the triggers that initiated the AF, however it failed to convert the AF to sinus rhythm. Following pulmonary vein (PV) angiography (Fig. 1A), two circular mapping catheters (Lasso, Biosense Webster, Diamond Bar, CA, USA) were placed in the superior and inferior PVs, respectively, and PV mapping was performed (Fig. 1B). The left PVs were active, and the potentials were fractionatedwith themean cycle length in the left PVs being very short (60–100 ms). The potentials in the right PVs were organized and the cycle length was longer than that in the left PVs. During ongoing AF, the left-sided ipsilateral PVs, including their antrum, were circumferentially ablated and isolated simultaneously under fluoroscopic and electrophysiologic guidancewith an 8-mm-tip ablation catheter (Japan Lifeline Inc., Tokyo, Japan) while monitoring the esophageal temperature (Fig. 1A and B). Following the isolation of the left PVs, the AF was easily converted to sinus rhythm with a 10 J" @default.
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- W2108422911 date "2011-09-01" @default.
- W2108422911 modified "2023-09-26" @default.
- W2108422911 title "Atrial fibrillation resistant to electrical cardioversion" @default.
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- W2108422911 doi "https://doi.org/10.1016/j.ijcard.2010.04.076" @default.
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