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- W2108497943 abstract "With the march of time, we all are slowly losing cardiomyocytes that are replaced by fibrocytes and collagen. This aging process likely occurs at different rates in different people and is influenced by concomitant disease, such as heart failure and hypertension with activation of the renin-angiotensin-aldosterone system. 1 Boldt A. Wetzel U. Lauschke J. Weigl J. Gummert J. Hindricks G. Kottkamp H. Dhein S. Fibrosis in left atrial tissue of patients with atrial fibrillation with and without underlying mitral valve disease. Heart. 2004; 90: 400-405 Crossref PubMed Scopus (373) Google Scholar , 2 Corradi D. Callegari S. Benussi S. Maestri R. Pastori P. Nascimbene S. Bosio S. Dorigo E. Grassani C. Rusconi R. Vettori M.V. Alinovi R. Astorri E. Pappone C. Alfieri O. Myocyte changes and their left atrial distribution in patients with chronic atrial fibrillation related to mitral valve disease. Hum Pathol. 2005; 36: 1080-1089 Abstract Full Text Full Text PDF PubMed Scopus (90) Google Scholar , 3 Healey J.S. Baranchuk A. Crystal E. Morillo C.A. Garfinkle M. Yusuf S. Connolly S.J. Prevention of atrial fibrillation with angiotensin-converting enzyme inhibitors and angiotensin receptor blockers: a meta-analysis. J Am Coll Cardiol. 2005; 45: 1832-1839 Abstract Full Text Full Text PDF PubMed Scopus (696) Google Scholar" @default.
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- W2108497943 date "2007-02-01" @default.
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- W2108497943 title "Substrate mapping and the aging atrium" @default.
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- W2108497943 doi "https://doi.org/10.1016/j.hrthm.2006.11.007" @default.
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