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• W2108601997 abstract "Sustained hypoxia (SH) has been shown to cause profound morphological and cellular changes in carotid body (CB). However, results regarding whether SH causes CB type I cell proliferation are conflicting. By using bromodeoxyuridine, a uridine analog that is stably incorporated into cells undergoing DNA synthesis, we have found that SH causes the type I cell proliferation in the CB; the proliferation occurs mainly during the first 1–3 days of hypoxic exposure. Moreover, the new cells survive for at least 1 mo after the return to normoxia. Also, SH does not cause any cell death in CB as examined by the terminal deoxynucleotidyl transferase-mediated dUTP-X nick-end labeling assay. Taken together, our results suggest that SH stimulates CB type I cell proliferation, which may produce long-lasting changes in CB morphology and function." @default.
• W2108601997 created "2016-06-24" @default.
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• W2108601997 date "2008-03-01" @default.
• W2108601997 modified "2023-10-16" @default.
• W2108601997 title "Sustained hypoxia-induced proliferation of carotid body type I cells in rats" @default.
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• W2108601997 doi "https://doi.org/10.1152/japplphysiol.00393.2007" @default.
• W2108601997 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/18096755" @default.
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