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- W2108706233 abstract "In 1989, Brown and colleagues [1] demonstrated that pretreatment of rats with a low dose of endotoxin (lipopolysaccharide, LPS) 24 hours prior to heart isolation and perfusion in the Langendorff mode resulted in an enhanced post-ischaemic recovery of myocardial function (left ventricular developed pressure, + dP/dt) and an increase in catalase activity. Hearts isolated from rats which were pretreated with LPS for only 1 hour did not have an increased catalase activity or functional protection from ischaemia-reperfusion injury. These results suggested that the delayed cardioprotection produced by LPS was the result of enhanced antioxidant enzyme activity, particularly catalase." @default.
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- W2108706233 title "Endotoxin, Monophosphoryl Lipid A and Delayed Cardioprotection" @default.
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