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- W2109115943 endingPage "130" @default.
- W2109115943 startingPage "130" @default.
- W2109115943 abstract "During the past decade a unifying hypothesis has been developed to explain the vascular changes that occur in septic shock on the basis of the effect of inflammatory mediators on the vascular endothelium. The vascular endothelium plays a central role in the control of microvascular flow, and it has been proposed that widespread vascular endothelial activation, dysfunction and eventually injury occurs in septic shock, ultimately resulting in multiorgan failure. This has been characterized in various models of experimental septic shock. Now, direct and indirect evidence for endothelial cell alteration in humans during septic shock is emerging. The present review details recently published literature on this rapidly evolving topic." @default.
- W2109115943 created "2016-06-24" @default.
- W2109115943 creator A5037085143 @default.
- W2109115943 date "2003-01-01" @default.
- W2109115943 modified "2023-09-25" @default.
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- W2109115943 doi "https://doi.org/10.1186/cc1864" @default.
- W2109115943 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/270612" @default.
- W2109115943 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/12720559" @default.
- W2109115943 hasPublicationYear "2003" @default.
- W2109115943 type Work @default.