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- W2109371472 endingPage "723" @default.
- W2109371472 startingPage "713" @default.
- W2109371472 abstract "Nonalcoholic fatty liver disease (NAFLD), hepatic insulin resistance, and type 2 diabetes are all strongly associated and are all reaching epidemic proportions. Whether there is a causal link between NAFLD and hepatic insulin resistance is controversial. This review will discuss recent studies in both humans and animal models of NAFLD that have implicated increases in hepatic diacylglycerol (DAG) content leading to activation of novel protein kinase Cϵ (PKCϵ) resulting in decreased insulin signaling in the pathogenesis of NAFLD-associated hepatic insulin resistance and type 2 diabetes. The DAG-PKCϵ hypothesis can explain the occurrence of hepatic insulin resistance observed in most cases of NAFLD associated with obesity, lipodystrophy, and type 2 diabetes. (Hepatology 2014;59:713-723)" @default.
- W2109371472 created "2016-06-24" @default.
- W2109371472 creator A5013839934 @default.
- W2109371472 creator A5027916227 @default.
- W2109371472 date "2014-01-29" @default.
- W2109371472 modified "2023-10-16" @default.
- W2109371472 title "Nonalcoholic fatty liver disease, hepatic insulin resistance, and type 2 Diabetes" @default.
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