Matches in SemOpenAlex for { <https://semopenalex.org/work/W2109555634> ?p ?o ?g. }
- W2109555634 abstract "The cardiac extracellular matrix (ECM) fills the space between cells, supports tissue organization, and transduces mechanical, chemical, and biological signals to regulate homeostasis of the left ventricle (LV). Following myocardial infarction (MI), a multitude of ECM proteins are synthesized to replace myocyte loss and form a reparative scar. Activated fibroblasts (myofibroblasts) are the primary source of ECM proteins, thus playing a key role in cardiac repair. A balanced turnover of ECM through regulation of synthesis by myofibroblasts and degradation by matrix metalloproteinases (MMPs) is critical for proper scar formation. In this review, we summarize the current literature on the roles of myofibroblasts, MMPs, and ECM proteins in MI-induced LV remodeling. In addition, we discuss future research directions that are needed to further elucidate the molecular mechanisms of ECM actions to optimize cardiac repair." @default.
- W2109555634 created "2016-06-24" @default.
- W2109555634 creator A5003454740 @default.
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- W2109555634 creator A5059824841 @default.
- W2109555634 creator A5090245008 @default.
- W2109555634 date "2014-02-13" @default.
- W2109555634 modified "2023-10-16" @default.
- W2109555634 title "Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodeling" @default.
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