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- W2110478085 abstract "A series of phorbol esters stimulates prostaglandin E production from both resident and starch-induced mouse peritoneal macrophages. The relative potencies of these compounds parallel their abilities both to elevate macrophage plasminogen activator levels and to function as tumor promoters in a mouse skin carcinogenesis model. Low concentrations of antiinflammatory glucocorticoids, such as dexamethasone, inhibit the enhanced prostaglandin E synthesis as a result of 12-O-tetradecanoylphorbol-13-acetate action. The same glucocorticoids also inhibit the enhanced plasminogen activator production. On the basis of these observations, it is suggested that there might be a relationship between macrophage phospholipase activation and plasminogen activator synthesis. It is also suggested that these data might be providing insight into how 12-O-tetradecanoylphorbol-13-acetate and certain structural analogs elicit an inflammatory response and behave as tumor promoters in mouse skin." @default.
- W2110478085 created "2016-06-24" @default.
- W2110478085 creator A5026903949 @default.
- W2110478085 date "1980-07-01" @default.
- W2110478085 modified "2023-10-18" @default.
- W2110478085 title "Stimulation of macrophage prostaglandin and neutral protease production by phorbol esters as a model for the induction of vascular changes associated with tumor promotion." @default.
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