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- W2110487265 endingPage "51" @default.
- W2110487265 startingPage "39" @default.
- W2110487265 abstract "Base excision repair (BER), as initiated by at least seven different DNA glycosylases or by enzymes that cleave DNA at abasic sites, executes the repair of a wide variety of DNA damages. Many of these damages arise spontaneously because DNA interacts with the cellular milieu, and so BER profoundly influences spontaneous mutation rates. In addition, BER provides significant protection against the toxic and mutagenic effects of DNA damaging agents present in the external environment, and as such is likely to prevent the adverse health effects of such agents. BER pathways have been studied in a wide variety of organisms (including yeasts) and here we review how these varied studies have shaped our current view of human BER." @default.
- W2110487265 created "2016-06-24" @default.
- W2110487265 creator A5047514569 @default.
- W2110487265 creator A5070554640 @default.
- W2110487265 date "2000-06-01" @default.
- W2110487265 modified "2023-10-18" @default.
- W2110487265 title "Base excision repair in yeast and mammals" @default.
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