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- W2110858029 endingPage "618" @default.
- W2110858029 startingPage "604" @default.
- W2110858029 abstract "Multiple sclerosis (MS) is an inflammatory and degenerative disease of the CNS with an assumed autoimmune-mediated pathogenesis. Stressful life events have been hypothesized as potential triggers of disease exacerbation. Animal studies using experimental autoimmune encephalomyelitis (EAE), as a model for MS, suggest that decreased hypothalamic–pituitary–adrenal (HPA) function may play a role in the increased susceptibility and severity of the disease. Histopathological studies of the hypothalamus point to disturbances in corticotropin-releasing hormone (CRH) regulation as a result of MS lesions in this area. Functional endocrine tests (e.g., the combined Dexamethasone–CRH test) showed a disturbed negative feedback after steroid application in MS patients. Hyper- and hypoactivity of the HPA axis, have been described to be associated with more severe courses. This paper presents an overview of the evidence for a role of HPA dysfunction in EAE and MS based on stress-experimental studies." @default.
- W2110858029 created "2016-06-24" @default.
- W2110858029 creator A5005739479 @default.
- W2110858029 creator A5015256073 @default.
- W2110858029 creator A5027159489 @default.
- W2110858029 creator A5044770070 @default.
- W2110858029 date "2007-07-01" @default.
- W2110858029 modified "2023-10-16" @default.
- W2110858029 title "Stress and hypothalamic–pituitary–adrenal axis function in experimental autoimmune encephalomyelitis and multiple sclerosis—A review" @default.
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