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- W2111414513 abstract "Calcitriol, a calcitrophic hormone that can be suppressed by high dietary calcium, favors fatty acid synthesis and inhibits lipolysis via non-genomic modulation of Ca(2+) influx. Calcitriol also suppresses UCP2 expression via the nVDR and thereby increases energy efficiency. Calcitriol exerts a dose-dependent impact on adipocyte apoptosis and regulates adipose tissue fat depot location and expansion by promoting glucocorticoid production and release. Recent data also demonstrate a pivotal role of calcitriol in the modulation of cytokines, with potential roles in energy metabolism in adipocytes, macrophages, and skeletal muscle." @default.
- W2111414513 created "2016-06-24" @default.
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- W2111414513 date "2008-09-25" @default.
- W2111414513 modified "2023-09-27" @default.
- W2111414513 title "Calcitriol and energy metabolism" @default.
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- W2111414513 doi "https://doi.org/10.1111/j.1753-4887.2008.00099.x" @default.
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