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• W2113250570 abstract "A human neuroblastoma cell line, IMR-32, was used as an in vitro model system to study the effects of arsenic trioxide (As2O3) on aggressive human neuroblastoma. From 0.5 μM, As2O3 exhibited a dose-dependent inhibition of IMR-32 proliferation. At concentrations of 1.5 μM or higher, As2O3 up-regulated caspase 3, leading to cellular apoptosis. However, neurofilament-200 kDa and tyrosine hydroxylase were not up-regulated, implying minimal neuronal differentiation. Concomitantly, TrkA was down-regulated and TrkB up-regulated. Pre-treatment with the protein kinase C (PKC) inhibitor Ro-31-8220 partially blocked As2O3-mediated apoptosis, meaning that As2O3 might signal through PKC activation. The results suggest that As2O3 might be potentially useful in neuroblastoma." @default.
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• W2113250570 date "2007-02-01" @default.
• W2113250570 modified "2023-10-01" @default.
• W2113250570 title "Effects of arsenic trioxide on the cellular proliferation, apoptosis and differentiation of human neuroblastoma cells" @default.
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• W2113250570 doi "https://doi.org/10.1016/j.canlet.2006.02.009" @default.
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