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- W2113299316 endingPage "591" @default.
- W2113299316 startingPage "573" @default.
- W2113299316 abstract "Childhood myeloid leukaemias are a diverse collection of conditions. Although many are also seen in adults, some are peculiar to childhood. In childhood AML, as in adults, cytogenetic abnormalities are associated with specific clinical features and define prognostic groups. In infants under 1 year with AML, the incidence of 11q23 abnormalities is particularly high. The finding of identical 11q23 breakpoints in infant leukaemia as in therapy-related leukaemias suggests a role for in utero exposure to topoisomerase II inhibitors. There are a number of constitutional disorders that predispose children to develop AML, usually with a preceding myelodysplastic phase. Monosomy (or deletion of the long arm) of chromosome 7 is the most frequent chromosome abnormality in the bone marrow of such patients. Abnormalities of chromosome 7 are also common cytogenetic findings in all morphological subgroups of childhood myelodysplasia, either as a primary abnormality or associated with disease progression." @default.
- W2113299316 created "2016-06-24" @default.
- W2113299316 creator A5088502139 @default.
- W2113299316 date "2001-09-01" @default.
- W2113299316 modified "2023-10-12" @default.
- W2113299316 title "Childhood myeloid leukaemias" @default.
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- W2113299316 doi "https://doi.org/10.1053/beha.2001.0155" @default.
- W2113299316 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11640870" @default.
- W2113299316 hasPublicationYear "2001" @default.
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