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- W2113449087 endingPage "127" @default.
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- W2113449087 abstract "The pathogenetic mechanisms underlying common, and less common but severe, adverse cutaneous drug reactions are reviewed. Pharmacogenetic variability may account for a susceptibility to serious drug reactions to sulphonamides and anticonvulsants, as well as to lupus erythematosus (LE)-like syndrome. Exanthematous drug reactions may have an immunological basis. Cell mediated cutaneous drug reactions, including lichenoid reactions, LE-like syndrome, fixed drug eruption, erythema multiforme, Stevens-Johnson syndrome and toxic epidermal necrolysis, will inevitably involve elements of the skin immune system. Graft-versus-host disease provides a useful model for aspects of these drug-induced disorders. Urticaria, angioedema, anaphlyaxis and anaphylactoid reactions may involve Type I immunoglobulin (Ig)-mediated or Type III hypersensitivity, or may be caused by pharmacological, non-allergic means. Drug-induced vasculitis, serum sickness and the Arthus phenomenon are manifestations of the immune complex disease. Drug-induced pemphigus may involve immune dysregulation, but several thiol-containing drugs are able to cause antibody-independent acantholysis directly." @default.
- W2113449087 created "2016-06-24" @default.
- W2113449087 creator A5067702004 @default.
- W2113449087 date "1995-08-01" @default.
- W2113449087 modified "2023-09-23" @default.
- W2113449087 title "Mechanisms of drug eruptions: Part I" @default.
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- W2113449087 doi "https://doi.org/10.1111/j.1440-0960.1995.tb00953.x" @default.
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