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• W2113647961 abstract "To investigate the physiological mechanisms that explain the end-exercise decrease in oxygen uptake during strenuous constant-power exercise, we recruited eleven trained, track cyclists. On two separated days they performed 1) resting spirometric measures, followed by an incremental test on a cycle ergometer to determine the power output at and 2) an exhaustive isokinetic supramaximal cycling exercise (Tlimsupra) at 185 ± 24% of (i.e., 640.5 ± 50.8 W). During cycling exercise tests, , ventilation parameters, stroke volume (SV) and heart rate were continuously recorded. Furthermore, arterialised capillary blood samples were collected to measure blood pH, arterial oxygen saturation, lactate and bicarbonate concentration before and 5 min after Tlimsupra. A > 5% decrease in and/or SV was observed in 6 subjects, with 5 out of 6 subjects presenting both phenomena. The magnitude of the decrease was correlated with the magnitude of the SV decrease (R = 0.75, P < 0.01), the peak-exercise end-tidal O2 partial pressure (R = 0.80, P < 0.005) and the resting, forced expiratory volume in 1 s (R = 0.72, P < 0.05), but not with any blood variables. The significant post-Tlimsupra decrease in forced vital capacity and forced inspiratory volume corroborate with a possible respiratory muscle fatigue. Based on these findings, we demonstrate that the occurrence of decrease in more than half of our subjects, during a strenuous constant-power exercise leading to a mild-acidosis (pH = 7.21 ± 0.04), results mainly from cardio-respiratory factors and not from blood metabolic responses." @default.
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• W2113647961 date "2013-12-01" @default.
• W2113647961 modified "2023-10-18" @default.
• W2113647961 title "Prevalence of cardio-respiratory factors in the occurrence of the decrease in oxygen uptake during supra-maximal, constant-power exercise" @default.
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• W2113647961 doi "https://doi.org/10.1186/2193-1801-2-651" @default.
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