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• W2113703021 abstract "Imatinib mesylate (IM) binds to the BCR-ABL protein, inhibiting its kinase activity and effectively controlling diseases driven by this kinase. IM resistance has been associated with kinase mutations or increased BCR-ABL expression. However, disease progression may be mediated by other mechanisms that render tumor cells independent of BCR-ABL. To demonstrate this potential, IM-resistant cells were found in chronic myelogenous leukemia patients with continuous BCR-ABL gene expression but undetectable BCR-ABL protein expression. These cells were unresponsive to IM and acquired BCR-ABL-independent signaling characteristics. IM resistance in some patients may be mediated through loss of kinase target dependence." @default.
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• W2113703021 date "2004-01-15" @default.
• W2113703021 modified "2023-09-26" @default.
• W2113703021 title "Imatinib Mesylate Resistance Through BCR-ABL Independence in Chronic Myelogenous Leukemia" @default.
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• W2113703021 doi "https://doi.org/10.1158/0008-5472.can-03-1484" @default.
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