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• W2114569908 abstract "Theoretical body-surface potentials were computed from single, branching and tortuous strands of Luo-Rudy dynamic model cells, representing different areas of an infarct scar. When action potential (AP) propagation either in longitudinal or transverse direction was slow (3-12 cm/s), the depolarization signals contained high-frequency (100-300 Hz) oscillations. The frequencies were related to macroscopic propagation velocity and strand architecture by simple formulas. Next, we extended a mathematical model of the QRS-complex presented in our earlier work to simulate unstable activation wavefront. It combines signals from different strands with small timing fluctuations relative to a large repetitive QRS-like waveform and can account for dynamic changes of real arrhythmogenic micropotentials. Variance spectrum of wavelet coefficients calculated from the composite QRS-complex contained the high frequencies of the individual abnormal signals. We conclude that slow AP propagation through fibrotic regions after myocardial infarction is a source of high-frequency arrhythmogenic components that increase beat-to-beat variability of the QRS, and wavelet variance parameters can be used for ventricular tachycardia risk assessment." @default.
• W2114569908 created "2016-06-24" @default.
• W2114569908 creator A5029014468 @default.
• W2114569908 creator A5080347335 @default.
• W2114569908 date "2004-11-01" @default.
• W2114569908 modified "2023-09-27" @default.
• W2114569908 title "A Theoretical Model of the High-Frequency Arrhythmogenic Depolarization Signal Following Myocardial Infarction" @default.
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• W2114569908 doi "https://doi.org/10.1109/tbme.2004.834277" @default.
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