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- W2115093094 abstract "Professor; Department of Anesthesiology; Massachusetts General Hospital; Harvard Medical School; Boston, Massachusetts 02114 (Martyn)Professor; Neurosciences Group; Institute of Molecular Medicine; John Radcliffe Hospital; University of Oxford; Oxford OX3 9DS, United Kingdom (Vincent)This Editorial View accompanies the following article: Tsukagoshi H, Morita T, Takahashi K, Kunimoto F, Goto F: Cecal ligation and puncture peritonitis model shows decreased nicotinic acetylcholine receptor numbers in rat muscle: Immunopathologic mechanisms? Anesthesiology 1999; 91:448–60.SEPSIS, with its associated complications, is a leading cause of death in intensive care units, with a mortality rate exceeding 40%. [1] An important untoward effect of such a critical illness is muscle wasting and muscle weakness, resulting in ventilator dependence with increasing morbidity and mortality rates. [2] The origin of these functional changes can be broadly classified as neural, neuromuscular (junctional), and/or muscular, [3,4] and these critically ill patients, particularly of the" @default.
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- W2115093094 title "A New Twist to Myopathy of Critical Illness " @default.
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- W2115093094 doi "https://doi.org/10.1097/00000542-199908000-00003" @default.
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