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• W2115965111 abstract "Abstract Introduction The present study aims to detect the specific and common impairment index relative to evoked brain potentials ( EBP S ) in deficit schizophrenia ( DS ) and nondeficit schizophrenia ( NDS ), and investigates the relationship between EBPs and clinical variables. Methods The study used EBPs in 21 patients with DS , 38 patients with NDS , and 50 healthy controls ( HCs ) to investigate P 300 waves, mismatch negative ( MMN ), sensory gating ( SG ) P 50 and contingent negative variation ( CNV ). A comparison of three groups and the relationship between EBPs and clinical variables were performed using general linear model analyses and partial correlation, respectively. Results Compared with HCs , both groups of patients showed delayed N 1 , N 2 , and P 3a latency, and reduced N 1 and N 2 amplitude. The MMN showed delayed latency. The P 50 ratios and the inhibited ratios were impaired, whereas SG loss ratios increased. CNV amplitude was reduced. Compared with HCs , NDS showed delayed latency of S 2′‐ C in CNV , whereas DS showed shortened latency. Only NDS , when compared with HCs , showed delayed latency of P 3b , Also, only DS , when compared with HCs , showed delayed latency of point A in CNV . Latency of point A in CNV of DS , correlated with a poorer G lobal A ssessment of F unctioning S cale (6 weeks) and was independent of clinical characteristics. Discussion Schizophrenia represents a clinical syndrome with shared impairments in brain function, whereas DS is a relatively homogeneous subgroup of schizophrenia with unique pathophysiological changes." @default.
• W2115965111 created "2016-06-24" @default.
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• W2115965111 date "2013-03-12" @default.
• W2115965111 modified "2023-10-17" @default.
• W2115965111 title "Multi-components of evoked-brain potentials in deficit and nondeficit schizophrenia" @default.
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• W2115965111 doi "https://doi.org/10.1111/appy.12030" @default.
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