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• W2116082009 abstract "Although autism spectrum conditions (ASCs) are defined behaviorally, they also involve multileveled disturbances of underlying biology that find striking parallels in the physiological impacts of electromagnetic frequency and radiofrequency exposures (EMF/RFR). Part I of this paper will review the critical contributions pathophysiology may make to the etiology, pathogenesis and ongoing generation of core features of ASCs. We will review pathophysiological damage to core cellular processes that are associated both with ASCs and with biological effects of EMF/RFR exposures that contribute to chronically disrupted homeostasis. Many studies of people with ASCs have identified oxidative stress and evidence of free radical damage, cellular stress proteins, and deficiencies of antioxidants such as glutathione. Elevated intracellular calcium in ASCs may be due to genetics or may be downstream of inflammation or environmental exposures. Cell membrane lipids may be peroxidized, mitochondria may be dysfunctional, and various kinds of immune system disturbances are common. Brain oxidative stress and inflammation as well as measures consistent with blood–brain barrier and brain perfusion compromise have been documented. Part II of this paper will review how behaviors in ASCs may emerge from alterations of electrophysiological oscillatory synchronization, how EMF/RFR could contribute to these by de-tuning the organism, and policy implications of these vulnerabilities. Changes in brain and autonomic nervous system electrophysiological function and sensory processing predominate, seizures are common, and sleep disruption is close to universal. All of these phenomena also occur with EMF/RFR exposure that can add to system overload (‘allostatic load’) in ASCs by increasing risk, and worsening challenging biological problems and symptoms; conversely, reducing exposure might ameliorate symptoms of ASCs by reducing obstruction of physiological repair. Various vital but vulnerable mechanisms such as calcium channels may be disrupted by environmental agents, various genes associated with autism or the interaction of both. With dramatic increases in reported ASCs that are coincident in time with the deployment of wireless technologies, we need aggressive investigation of potential ASC – EMF/RFR links. The evidence is sufficient to warrant new public exposure standards benchmarked to low-intensity (non-thermal) exposure levels now known to be biologically disruptive, and strong, interim precautionary practices are advocated." @default.
• W2116082009 created "2016-06-24" @default.
• W2116082009 creator A5003268257 @default.
• W2116082009 creator A5074539208 @default.
• W2116082009 date "2013-06-01" @default.
• W2116082009 modified "2023-09-27" @default.
• W2116082009 title "Autism and EMF? Plausibility of a pathophysiological link – Part I" @default.
• W2116082009 cites W1506616055 @default.
• W2116082009 cites W1520936573 @default.
• W2116082009 cites W156121289 @default.
• W2116082009 cites W1576097100 @default.
• W2116082009 cites W1652297607 @default.
• W2116082009 cites W1813562443 @default.
• W2116082009 cites W1878727079 @default.
• W2116082009 cites W1905863734 @default.
• W2116082009 cites W1964444099 @default.
• W2116082009 cites W1965962074 @default.
• W2116082009 cites W1967357002 @default.
• W2116082009 cites W1967564742 @default.
• W2116082009 cites W1969459785 @default.
• W2116082009 cites W1969926229 @default.
• W2116082009 cites W1970467499 @default.
• W2116082009 cites W1970660523 @default.
• W2116082009 cites W1970724586 @default.
• W2116082009 cites W1970755818 @default.
• W2116082009 cites W1970848271 @default.
• W2116082009 cites W1971159934 @default.
• W2116082009 cites W1971757569 @default.
• W2116082009 cites W1972479743 @default.
• W2116082009 cites W1976383834 @default.
• W2116082009 cites W1976388899 @default.
• W2116082009 cites W1978222253 @default.
• W2116082009 cites W1978357172 @default.
• W2116082009 cites W1978992015 @default.
• W2116082009 cites W1979050871 @default.
• W2116082009 cites W1979070592 @default.
• W2116082009 cites W1979375310 @default.
• W2116082009 cites W1979499161 @default.
• W2116082009 cites W1979624408 @default.
• W2116082009 cites W1980505500 @default.
• W2116082009 cites W1981271928 @default.
• W2116082009 cites W1982718433 @default.
• W2116082009 cites W1983435780 @default.
• W2116082009 cites W1984551174 @default.
• W2116082009 cites W1984947171 @default.
• W2116082009 cites W1985944382 @default.
• W2116082009 cites W1986167717 @default.
• W2116082009 cites W1988372841 @default.
• W2116082009 cites W1989415566 @default.
• W2116082009 cites W1989421328 @default.
• W2116082009 cites W1989779456 @default.
• W2116082009 cites W1990681224 @default.
• W2116082009 cites W1991979585 @default.
• W2116082009 cites W1992584198 @default.
• W2116082009 cites W1992629138 @default.
• W2116082009 cites W1992758695 @default.
• W2116082009 cites W1993636611 @default.
• W2116082009 cites W1993752158 @default.
• W2116082009 cites W1994710518 @default.
• W2116082009 cites W1994983237 @default.
• W2116082009 cites W1995647101 @default.
• W2116082009 cites W1997647948 @default.
• W2116082009 cites W1998046661 @default.
• W2116082009 cites W2000262719 @default.
• W2116082009 cites W2000309815 @default.
• W2116082009 cites W2002428861 @default.
• W2116082009 cites W2002518831 @default.
• W2116082009 cites W2003018284 @default.
• W2116082009 cites W2004279679 @default.
• W2116082009 cites W2004575036 @default.
• W2116082009 cites W2004961725 @default.
• W2116082009 cites W2005356339 @default.
• W2116082009 cites W2007005070 @default.
• W2116082009 cites W2007094743 @default.
• W2116082009 cites W2007593843 @default.
• W2116082009 cites W2008041264 @default.
• W2116082009 cites W2008466227 @default.
• W2116082009 cites W2008578046 @default.
• W2116082009 cites W2010915134 @default.
• W2116082009 cites W2012860479 @default.
• W2116082009 cites W2014797499 @default.
• W2116082009 cites W2017048113 @default.
• W2116082009 cites W2018871086 @default.
• W2116082009 cites W2020407498 @default.
• W2116082009 cites W2020523926 @default.
• W2116082009 cites W2020727079 @default.
• W2116082009 cites W2023239280 @default.
• W2116082009 cites W2023803903 @default.
• W2116082009 cites W2024563625 @default.
• W2116082009 cites W2024663595 @default.
• W2116082009 cites W2025306107 @default.
• W2116082009 cites W2026643647 @default.
• W2116082009 cites W2028878165 @default.
• W2116082009 cites W2030462908 @default.
• W2116082009 cites W2030765708 @default.
• W2116082009 cites W2030789747 @default.
• W2116082009 cites W2032011914 @default.
• W2116082009 cites W2032647396 @default.

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