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- W2116716023 endingPage "200" @default.
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- W2116716023 abstract "Atherosclerosis and its complications, such as coronary heart disease, heart infarction and stroke, are the leading causes of death in the developed world. High blood pressure, diabetes, smoking and a diet high in cholesterol and lipids clearly increase the likelihood of premature atherosclerosis, albeit other factors, such as the individual genetic makeup, may play an additional role. During atherosclerosis, uncontrolled cholesterol and lipid accumulation in macrophages and smooth muscle cells leads to foam cell formation and to the progression of the atherosclerotic plaque. This review will focus on foam cell formation within the atherosclerotic lesion, the involvement of the scavenger receptor genes in this process, and the possibility to interfere with scavenger receptor function to reduce the progression of atherosclerosis. To date, the regulatory mechanisms for the expression of scavenger receptor genes and their role in atherosclerosis are not well characterized. Knowledge on this subject could lead to a better understanding of the process, prevention and therapy of this disease." @default.
- W2116716023 created "2016-06-24" @default.
- W2116716023 creator A5042840198 @default.
- W2116716023 creator A5069545876 @default.
- W2116716023 creator A5075026169 @default.
- W2116716023 date "2000-01-01" @default.
- W2116716023 modified "2023-10-16" @default.
- W2116716023 title "Scavenger receptor regulation and atherosclerosis" @default.
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- W2116716023 doi "https://doi.org/10.1002/biof.5520110305" @default.
- W2116716023 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/10875306" @default.
- W2116716023 hasPublicationYear "2000" @default.
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