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• W2116765932 abstract "To establish models for studying recurrence of visceral leishmaniasis, a growing problem in T cell-deficient patients, two approaches were investigated: treatment of euthymic BALB/c mice with quiescent Leishmania donovani infection with T cell-depleting or anti-cytokine antibodies and serial observation of acutely infected nude BALB/c mice after an initial antileishmanial response induced by amphotericin B treatment. In chronically infected euthymic mice, maintenance of acquired immunity and prevention of relapse required CD4 cells and a multicytokine-dependent mechanism involving endogenous interleukin-2, interferon-γ, and tumor necrosis factor-α. Acutely infected nude mice responded to amphotericin B with a ⩾85% reduction in liver parasite burdens; however, after a brief lag, visceral infection readily recurred in the posttreatment period. Both models may be useful for testing experimental interventions designed to reduce relapse of previously controlled visceral leishmaniasis in T cell-deficient hosts." @default.
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• W2116765932 date "1996-04-01" @default.
• W2116765932 modified "2023-09-26" @default.
• W2116765932 title "Models of Relapse of Experimental Visceral Leishmaniasis" @default.
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• W2116765932 doi "https://doi.org/10.1093/infdis/173.4.1041" @default.
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