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- W2116892457 endingPage "13191" @default.
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- W2116892457 abstract "Spinal cord injury (SCI) results in neuronal and glial death and the loss of axons at the injury site. Inflammation after SCI leads to the inhibition of tissue regeneration and reduced neuronal survival. In addition, the loss of axons after SCI results in functional loss below the site of injury accompanied by neuronal cell body’s damage. Consequently, reducing inflammation and promoting axonal regeneration after SCI is a worthy therapeutic goal. The receptor for advanced glycation end products (RAGE) is a transmembrane protein and receptor of the immunoglobulin superfamily. RAGE is implicated in inflammation and neurodegeneration. Several recent studies demonstrated an association between RAGE and central nervous system disorders through various mechanisms. However, the relationship between RAGE and SCI has not been shown. It is imperative to elucidate the association between RAGE and SCI, considering that RAGE relates to inflammation and axonal degeneration following SCI. Hence, the present review highlights recent research regarding RAGE as a compelling target for the treatment of SCI." @default.
- W2116892457 created "2016-06-24" @default.
- W2116892457 creator A5006006806 @default.
- W2116892457 creator A5017475311 @default.
- W2116892457 creator A5051275961 @default.
- W2116892457 creator A5057470890 @default.
- W2116892457 date "2014-07-25" @default.
- W2116892457 modified "2023-10-12" @default.
- W2116892457 title "Receptor for Advanced Glycation End Products (RAGE) and Its Ligands: Focus on Spinal Cord Injury" @default.
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